Sullivan mitochondrias ketogenic diet

By | August 26, 2020

sullivan mitochondrias ketogenic diet

Recently, it was shown that the high alkaline pH of the mitochondria in the presence of elevated levels of acetyl-CoA allows for nonenzymatic acetylation to occur in vitro Enhanced flux of acetyl-CoA from ATP-citrate lyase increased acetylation of H3 and this resulted in increased expression of glycolytic enzymes. A modified Atkins diet is effective for the treatment of intractable pediatric epilepsy. Houtkooper R. Barja and A. Desquiret-Dumas et al. Dai, Y. Kim et al. As another component of the NT theory, it was hypothesized that the KD might also enhance reuptake of glutamate from the synaptic cleft by astrocytes Elevated polyunsaturated fatty acids in blood serum obtained from children on the ketogenic diet. Kim, K.

Read article at publisher’s site DOI : Mattson MP, Liu D. Nutrients, 12 6, 17 Jun Int J Mol Sci, 21 9, 05 May Nutrients, 12 4, 08 Apr Nutrients, 12 3, 17 Mar To arrive at the top five similar articles we use a word-weighted algorithm to compare words from the Title and Abstract of each citation. Nature, , 01 Jan Cited by: articles PMID:

Hence, it follows that metabolism of ketone bodies may circumvent this deficit and allow for oxidative phosphorylation to occur, possibly by enhancing the activity of complex II in the ETC 3. Norris, and K. Klingenberg and K. Veech R. Efficacy of the Atkins diet as therapy for intractable epilepsy. Rockl, M. Mattingly, R. Mady M. Hirschey et al.

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